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Elucidating the exact pharmacological mechanism of action (MOA) of naturally happening compounds might be demanding. Despite the fact that Tarselli et al. (60) developed the main de novo artificial pathway to conolidine and showcased that this naturally developing compound correctly suppresses responses to both of those chemically induced and infla

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Preliminary scientific tests point out conolidine might inhibit precise ion channels, cutting down neuronal excitability and limiting agony indicators. This mechanism is particularly applicable in neuropathic soreness, in which abnormal signaling triggers persistent discomfort. Also, conolidine seems to influence G protein-coupled receptor (GPCR) p